Download e-book for kindle: Alcohol Related Diseases in Gastroenterology by W. K. Lelbach (auth.), Priv. Doz. Dr. med. Helmut Karl

By W. K. Lelbach (auth.), Priv. Doz. Dr. med. Helmut Karl Seitz, Professor Dr. med. Burkhard Kommerell (eds.)

ISBN-10: 3642700489

ISBN-13: 9783642700484

ISBN-10: 3642700500

ISBN-13: 9783642700507

Alcohol abuse ranks one of the commonest and in addition the main critical environmental dangers to human healthiness. Its value is heightened via the opportunity of prevention via removal of the behavior, notwithstanding, hardly ever exerted. The occurrence of deleterious results on human future health has relentlessly risen long ago years for various elements. They comprise migration of populations and, fairly, elevated urbanization. hence, in a few components of the area, inhabitants teams formerly spared became concerned, that's additionally re­ flected within the expanding variety of breweries and distilleries within the constructing nations. Social, non secular, and gender-related boundaries to alcohol intake are loosening, and the monetary development of a few segments of populations now let them to shop for alcoholic drinks. therefore the best percent upward thrust within the usa has lately been in black ladies. kids and youth drink extra alcoholic drinks than ever, and turning out to be alcohol abuse via pregnant ladies has allow to a rise of the occurrence of the fetal alcohol syndrome. whereas the social and behavioral, together with psychiatric, results of alcoholism are impressive, the gastrointestinal and, quite, hepatic manifestations are the main common somatic results, and persistent hepatic affliction in alcoholics seems to reason the best rate to society. certainly, mortality from liver cirrhosis is taken into account a competent index of alcohol intake in a country.

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Gastroenterology 66: 415-422 Hasumura Y, Teschke R, Lieber CS (1975a) Hepatic microsomal ethanol oxidizing system (MEOS): dissociation from reduced nicotinamide adenine dinucleotide phosphate-oxidase and possible role of from I of cytochrome P-450. J Pharmacol Exp Ther 194: 469-474 Hasumura Y, Teschke R, Lieber CS (1975b) Acetaldehyde oxidation by hepatic mitochondria: its decrease after chronic ethanol consumption. Science 189: 727 -729 Hasumura Y, Teschke R, Lieber CS (1976) Characteristics of acetaldehyde oxidation in rat liver mitochondria.

Res Exp Med (Bed) 163: 1-16 Gordon ER (1973) Mitochondrial functions in an ethanol-induced fatty liver. J Bioi Chem 248: 8271-8280 Gordon ER (1977) ATP metabolism in an ethanol induced fatty liver. Alcoholism 1: 21-5 Gordon ER, Rochman J, Arai M, Lieber CS (1982) Lack of correlation between hepatic mitochondrial membrane structure and functions in ethanol-fed rats. Science 216: 1319-1321 Green H, Goldberg B (1964) Collagen and cell protein synthesis by an established mammalian fibroblast line. Nature 204: 347-349 Gregory DH, Vlahcevic ZR, Prugh MF, Swell L (1978) Mechanism of secretion of biliary lipids: role of a microtubular system in hepatocellular transport of biliary lipids in the rat.

1974). It is likely that a similar mechanism increases susceptibility in these patients to the hepatotoxic actions of a variety of compounds in the environment, including many commonly used drugs such as ioniazid and acetaminophen (Sato et al. 1981), and possibly carcinogens. These aspects are discussed in detail by Sato, Hasumura, and Seitz in other chapters. Enhanced microsomal degradation of endogenous substrates such as steroid hormones was also observed after chronic ethanol consumption; its impact on the endocrine status of the alcoholic is described by Van Thiel in this volume.

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Alcohol Related Diseases in Gastroenterology by W. K. Lelbach (auth.), Priv. Doz. Dr. med. Helmut Karl Seitz, Professor Dr. med. Burkhard Kommerell (eds.)

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